Category: Heat Shock Protein 90

Med

Med. 15:691C700 [PMC free article] [PubMed] [Google Scholar]. of phosphorylated -catenin (Ser552) as an EMT mediator, which translocated into the nucleus and triggered Akt. The phosphorylation level of -catenin at Thr41/Ser45 moieties was specifically higher in control than in HCV-infected hepatocytes, implicating an inactivation of -catenin. Collectively, these results suggested that primary human being hepatocytes […]

The necrotic areas were decreased by almost half upon EC-specific Nrf2 activation (Figure 3E)

The necrotic areas were decreased by almost half upon EC-specific Nrf2 activation (Figure 3E). protection in SCD. However, little is known regarding the mechanisms by which Nrf2 ameliorates SCD pathology or how some cells respond to Nrf2 stimuli to alleviate SCD pathology. Here, we asked whether monocytes/granulocytes and/or endothelial cells are particularly critical in alleviating […]